Discussion on GABA as a key player in mediating neuroimmune communications between NK cells and DC.

GEM, small GTPase from the RGK family, serves as significant negative regulator of neutrophils' activity.

In response to infection challenge, NK cells synthesize and secrete gamma-aminobutyric acid (GABA), which impacts NK cell and dendritic cell functions via GABA-A receptors.

Deficiency of the small GTPase Gem slows down the autophagic flux in stimulated neutrophils, causing autophagosome accumulation and autophagosome-associated NADPH complex assembling, leading to increased ROS and NETs formation.

LncRNA SNHG12 alleviates vascular endothelial injury in Ang II-induced hypertension through miR-25-3p/SIRT6 pathway.

Exogenous ATP enhances LPS-mediated PGE₂ release in macrophages by sustaining a high level of COX-2 mRNA through extended phosphorylation of CDK9 and p38 MAPK.

The flexibility of eosinophil gene expression profiles in vivo supports the LIAR hypothesis of eosinophil function.

IAPs regulate the survival of pro- and anti-inflammatory human macrophages and targeting the IAPs and/or caspases can be exploited to control inflammation-related disorders

Administration of adipose-derived MSCs expressing a dominant-negative HMGA1 transgene results in less tissue injury and inflammation during a systemic infection, while improving neutrophil phagocytosis.

Activation by anti-CD20 mAb upregulates NK cell TIGIT and TIM3 expression but has minimal effect on NK cell phenotype or function.

Comparing human and mouse PU.1 regulome and interactome in inflammatory DCs reveals species-specific responses and conserved regulatory features of PU.1.

Reviews the spleen's function during malaria infection; shows how this organ serves as a central hub for activating and exerting effector mechanisms within the immune response to control blood-stage infection with Plasmodium parasites.

Review comparing IL-34 and CSF-1, including their biochemistry, biology, and roles in diseases; highlighting new areas of research and potential therapies.

Review of mechanisms of neutrophil extracellular traps (NETs) formation, including the impact of NETs on individual organs as well as the limitations and advantages of NETs detection assays in use today.

Review on Vitamin D-dependent immunoregulatory mechanisms underlying respiratory disease severity in SARS-CoV2 infections.