Review of the mechanisms underlying the immune and inflammatory damage from keratinocytes caused by S. aureus; may help guide treatment options for patients with atopic dermatitis.

SNHG8 reduced microglia activation and blood-brain barrier permeability via the miR-449c-5p/SIRT1/SIRT1/FoxO1 pathway, thus playing a protective role in ischemic brain injury.

Examines the functional mechanism of CA in AM polarization in detail and provides a theoretical basis for treatment of Klebsiella pneumoniae (Kp)-induced pneumonia.

mTORC2-dependent Akt activation is instrumental for metabolic reprogramming at the early stages-migration and differentiation-of macrophage-mediated immunity.

Single cell transcriptomics of peripheral blood mononuclear cells in surviving and fatal sepsis reveal inflammatory and metabolic pathways that change within hours of sepsis recognition.

Review of current extracellular vesicle-based treatment modalities in hemostasis, inflammation, acute lung injury, traumatic brain injury, and spinal cord injury following trauma, and potential for the future.

BCG immune training before pregnancy led to IUGR in mice by modifying immune cell frequency in the developing placenta, whereas LPS training had no effect

Flunarizine limits Mycobacterium tuberculosis growth in macrophages by promoting CaM-pCAMKII-mediated phagolysosome maturation.

Reviews several essential and recently discovered signaling pathways in B cell development; takes a look at diseases related to aberrant signals as well as newly developed strategies for treating these diseases.

Review of the role of extracellular vesicles during the different phases of atherosclerotic lesion development that describes the need to standardize isolation and analysis procedures to fully realize their potential as biomarkers and therapeutics for cardiovascular diseases.

Identification of MFN2 as a key regulator of leukocyte adhesion, integrin activation, actin polymerization, and maturation.

Peony seed oil suppressed microglial activation and neuroinflammation through the downregulation of pro-inflammatory mediators in the prefrontal cortex and hippocampus of PS cDKO mice, and lessened memory impairment by reversing hyperphosphorylated tau and synaptic proteins deficits.

TNFR1 is pathogenic while TNFR2 plays a protective role in the development of psoriasis in the mouse model of psoriasis induced by imiquimod.

Review on the scientific premise for incidence of vaccine-induced thrombotic thrombocytopenia as a result of inflammatory reactions mediated via IL-1β/NLRP3 inflammasome activation and consequent overproduction of NETs.

Reviews helminth infection's impact on the immune system and the microbiota; provides mechanistic insight that may aid in finding new approaches for treating cardiovascular disease.